Studies

CellWell record HH-476841

Pathophysiological significance of impaired KAT7-dependent histone H3K14 acetylation during zinc deficiency

Takao Fujisawa; Satoshi Takenaka; Lila Maekawa; Motoyuki Ogawa; Toshiyuki Kowada; Toshitaka Matsui; Shin Mizukami; Yugo Kato; Michio Suzuki; Hisashi Noma; Isao Naguro; Hidenori Ichijo

Nature Communications ยท 2026

In plain language

Zinc deficiency inactivated the enzyme KAT7 and lowered H3K14 acetylation, which raised the zinc transporter ZIP10 in cells and was accompanied by liver lipid buildup in mice.

Evidence
Emerging
Design
Primary research (design unspecified)

Read it on PubMed

Powered by CellWell

Cite this record: https://thecellwell.org/studies/HH-476841

Produced by the CellWell Research Engine, custom software owned and operated by CellWell and The Holistic Hub. All rights reserved. This analysis, its indexing and its evidence classifications are proprietary. Any use, republication or onward sharing must credit CellWell Research Engine (thecellwell.org) as the source.

This is an educational research library, not medical advice. Nothing here may be used to diagnose, treat, cure, or prevent any disease. Always consult a qualified professional.