CellWell record HH-13271
Disorders of aldosterone synthesis, secretion, and cellular function.
Root AW
Current opinion in pediatrics ยท 2014
In plain language
Review describes primary and secondary hypoaldosteronism as arising from mutations in genes encoding the mineralocorticoid receptor, epithelial sodium channel subunits, or WNK-pathway regulators that disrupt renal tubular sodium, potassium, and chloride cotransport.
- Studied in
- Human
Cite this record: https://thecellwell.org/studies/HH-13271
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